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Ecological and epidemiological modeling is an important interdisciplinary research area that integrates ecological interactions with the spread of infectious diseases in biological populations. It aims to understand how diseases influence the dynamics of species such as prey, predators, hosts, and vectors within an ecosystem. Using mathematical tools such as differential equations, researchers analyze population growth, disease transmission, recovery, predation, competition, and environmental effects simultaneously. These models help explain complex phenomena including species persistence, extinction, oscillatory behavior, and outbreak thresholds. Ecological and epidemiological modeling has wide applications in wildlife conservation, pest management, livestock health, and public health planning, as it provides valuable insights for designing effective control and management strategies.
Chaos and bifurcation theory is a significant branch of nonlinear dynamical systems that studies sudden qualitative changes in system behavior and the emergence of complex irregular dynamics as parameters vary. Bifurcation theory explains how small changes in growth rates, transmission coefficients, harvesting levels, or interaction strengths can shift a system from stable equilibrium to periodic oscillations, multiple steady states, or instability. Chaos theory investigates deterministic systems that exhibit highly sensitive dependence on initial conditions, leading to unpredictable long-term behavior despite having fixed governing equations. In ecological and epidemiological models, these concepts are useful for understanding population cycles, disease outbreaks, extinction risks, and abrupt regime shifts. The analysis of chaos and bifurcations helps researchers identify critical thresholds and improve management and control strategies in complex biological systems.
·Delay differential equations are an important class of dynamical systems in which the rate of change of a variable depends not only on its present state but also on its past states. These equations are used to model processes where time delays naturally occur, such as gestation periods, maturation time, incubation delays, immune response lags, and resource regeneration. In ecological and epidemiological modeling, delay differential equations help describe more realistic population growth, predator-prey interactions, and disease transmission dynamics. The inclusion of delays can significantly affect system behavior by generating oscillations, instability, bifurcations, or sustained periodic solutions that may not appear in ordinary differential equation models. Therefore, delay differential equations are valuable tools for understanding memory effects and time-lagged responses in biological and environmental systems.
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Title: Role of alternative food in controlling chaotic dynamics in a predator–prey model with disease in the predator.
Title: Simultaneous Effects of Prey Defence and Predator Infection on a Predator Prey System.
Our current research is based on the investigation of an eco-epidemiological model that solely includes illness in predators. Predators, both healthy and diseased, are thought to consume prey and breed; however, the offsprings are expected to be vulnerable. To achieve a more realistic and explicit outcome of the existing phenomena correlated with our model system, we consider that the process of disease transmission is mediated by some time lag and the intensity of disease prevalence is seasonally forced. Our simulation results show that the disease dies out for lower intensity of disease prevalence or higher rate of consumption of prey by susceptible predator. The system undergoes subcritical/supercritical Hopf bifurcation due to changes in the parameters representing the intensity of disease prevalence, consumption rate of prey by susceptible/infected predator. The system exhibits two types of bistabilities: the first one between stable coexistence and oscillating coexistence, and the second one between two oscillatory coexistence cycles. Moreover, we see that with gradual increase in the incubation delay, the system shows transitions from stable focus to limit cycle oscillations to period doubling oscillations to chaotic dynamics. Chaotic dynamics is also observed for the periodic changes in the intensity of disease prevalence if it takes much time for the pathogens to develop sufficiently inside the body of the susceptible predator population.
In this paper, we propose and analyse a predator-prey model with disease in prey. We assume that a portion of healthy prey takes refuge to avoid predation. We find the biologically feasible equilibrium points and their stability criteria by using linearization technique. We also perform Hopf bifurcation analysis around the coexisting equilibrium point. We carry out extensive numerical simulation to validate our theoretical results and also explore rich dynamics which cannot be attained analytically. We draw some one and two parameter bifurcation diagrams which demonstrate rich dynamics like, Hopf bifurcation, chaos, bistability, etc. We observe that invasion of disease in prey can produce chaos through period-doubling bifurcation, whereas refuge can control chaos via period-halving bifurcation. We also observe that refuge can control disease prevalence in the prey population.
In this research work, the Rosenzweig-MacArthur model comprises with the predator population afflicted by illness has been studied. We may investigate the effects of Allee effects further by looking at populations of predators and prey with a weak Allee effect and prey with a strong Allee effect. We carry out analytical analyses of equilibrium, stability, and Hopf bifurcation. We have also investigated, via numerical simulations, chaotic dynamics for a progressive rise in the force of infection. We see that a strong Allee effect can keep the system from going into chaos, but that once the strong Allee parameter reaches a certain value, the prey population goes extinct and as a result, also the predator population extincts too. However, the weak Allee effect also has a important impact on population dynamics and has the potential to increase the number of predators. To investigate the complex dynamics of the system, we have created a number of bifurcation diagrams with one and two parameters. We also note that at appropriate parameter values, the system may display tri-stability and bi-stability.
We propose and analyze a three-dimensional eco-epidemiological model involving susceptible prey, infected prey, and predators, where the predators are supplemented with an externally supplied constant quantity of additional food. The model incorporates nonlinear disease transmission and predator feeding saturation through a generalized Holling type II functional response. We investigate the system’s dynamics analytically and numerically by studying the existence and stability of equilibria, as well as bifurcations including Hopf, transcritical, and saddle-node bifurcations. One and two parameter bifurcation analyses reveal rich dynamics such as limit cycles, period-doubling, and chaotic oscillations. Our findings indicate that disease transmission can destabilize the system, while the inclusion of additional food enhances stability and can suppress chaos. Furthermore, we extend the model by introducing a time-dependent optimal control variable representing additional food supply, and derive an optimal strategy using Pontryagin’s Maximum Principle. Numerical simulations show that the optimal control effectively reduces disease prevalence and stabilizes the population dynamics. This study highlights the potential of ecological interventions, such as strategic food supplementation, in regulating complex eco-epidemiological systems.
Eco-epidemiological systems, in which infectious diseases interact with ecological processes such as predation and competition, exhibit rich and often counterintuitive dynamics. In predator–prey systems where the prey population is subject to disease, additional ecological mechanisms such as the Allee effect and non-consumptive fear responses can critically influence stability, persistence, and extinction outcomes. Furthermore, biological processes like predator gestation introduce time delays that can fundamentally alter system trajectories. In this study, we develop and analyze a nonlinear predator–prey–disease model incorporating (i) a strong Allee effect in the prey population, (ii) fear-mediated reductions in prey growth rates, and (iii) an explicit gestation delay in predator reproduction. Analytical investigations are conducted to determine the existence and stability conditions of equilibria, extinction thresholds, and bifurcation structures. The delay is treated as a bifurcation parameter to assess its influence on the stability of the coexistence equilibrium and the onset of oscillatory dynamics. Numerical simulations further reveal delay-induced destabilization, bistability, and shifts in the basins of attraction. The results highlight how the combined effects of ecological constraints, behavioral adaptations, and epidemiological factors shape the qualitative dynamics of eco-epidemiological systems, offering new insights into population persistence and control strategies.
In this research work, an eco-epidemiological model comprises with disease in prey population and strong Allee effect also in prey population has been considered. In this work, we have also considered alternative prey as living prey population. We carry out the analytical analysis of equilibria and stability. We also investigated via numerical analysis, chaotic dynamics of the system for progressive increment of disease in prey population. We see that if strong Allee effect parameter crosses the certain value, the system settles down to stability. However, the alternative prey population has an important impact on population dynamics making the system stable first and then gradually disease free. To understand the complex dynamics of the system, we have also investigated different one parametric bifurcation and two parametric bifurcation. We have also noted that, for appropriate parameter values, the system shows different bi-stability in different regions.
In this work we have analyzed an eco-epidemic model with adaptive predation strategy in predator population. We have drawn average fitness function of foraging strategy and analyzed the stability of the system for two different cases. The subsystem shows chaotic dynamics for increasing infection ratethrough period doubling bifurcation. And also shows bi-stability between interior equilibrium and stable limit cycle. To show the rich dynamics of the system we have drawn two parametric bifurcation in different parametric space.
This study focuses on the dynamical behavior of a deterministic eco-epidemic model incorporating the Beddington-DeAngelis functional response and a fractional-order framework with an optimal control strategy. The model describes the interactions between prey and predator populations in the presence of infectious disease, where the Beddington-DeAngelis functional response accounts for predator interference and more realistic predation behavior. The use of fractional-order derivatives introduces memory and hereditary effects, making the system better suited to represent real biological processes. Qualitative analysis such as positivity, boundedness, equilibrium existence, local and global stability, bifurcation, and persistence can be investigated to understand the long-term dynamics of the system. Furthermore, optimal control techniques are applied to determine effective strategies such as treatment, vaccination, harvesting, or culling while minimizing economic and ecological costs. This approach provides deeper insight into disease management and ecosystem sustainability.
This study deals with the sensitivity and bifurcation analysis of an SEIR-type dengue disease model incorporating vaccination as a control measure. The population is divided into susceptible, exposed, infected, and recovered classes to describe the transmission dynamics of dengue fever. Vaccination is included to reduce susceptibility and limit the spread of infection within the community. Sensitivity analysis is performed to identify the most influential parameters affecting the basic reproduction number and disease prevalence, such as transmission rate, recovery rate, mosquito biting rate, and vaccine efficacy. Bifurcation analysis is used to examine qualitative changes in system behavior, including the possibility of backward or forward bifurcation, multiple endemic equilibria, and threshold dynamics near the critical reproduction number. This investigation provides important insights for designing efficient vaccination policies and public health strategies for dengue prevention and control.
This study considers a stage-structured prey-predator model with a Beddington–DeAngelis type functional response incorporating prey refuge. The prey population is divided into different life stages, such as juvenile and mature classes, to represent realistic growth and maturation processes. The Beddington–DeAngelis functional response is used to describe predation more accurately by including mutual interference among predators during hunting. A prey refuge is introduced to represent a proportion of prey protected from predation due to hiding places or environmental shelter. The model is used to investigate important dynamical properties such as positivity, boundedness, equilibrium existence, local and global stability, persistence, and bifurcation behavior. The inclusion of stage structure and refuge mechanisms provides deeper insight into species coexistence, predator survival, and ecological balance in natural ecosystems.
This study examines an ecological model of aposematism incorporating the Beddington–DeAngelis functional response and Shepherd’s recruitment function. Aposematism refers to the use of warning coloration or signals by prey species to reduce predation by indicating toxicity or unpalatability. The Beddington–DeAngelis functional response is employed to represent realistic predator-prey interactions by accounting for predator interference during the searching and handling process. Shepherd’s recruitment function is used to model density-dependent population recruitment, allowing flexible growth behavior under low and high population densities. The combined model can be analyzed to investigate key dynamical features such as equilibrium existence, stability, persistence, oscillatory dynamics, and bifurcation phenomena. This framework helps explain how warning signals, predator behavior, and population regulation jointly influence species survival and long-term ecological balance.
This study focuses on a plant-pest interaction model based on the Mango–Gall midge system, where mango plants serve as the host and gall midges act as destructive pests. Gall midges damage leaves, flowers, and developing tissues, reducing plant growth, fruit yield, and overall productivity. The mathematical model describes the population dynamics between mango plants and pest species by incorporating factors such as plant growth, pest infestation, reproduction, natural mortality, and possible control measures. Analysis of the system may include equilibrium points, stability behavior, persistence, threshold conditions, and bifurcation dynamics. The model can also be extended to include biological control, pesticide application, seasonal effects, or delay factors. Such studies are useful for understanding pest outbreaks and developing effective management strategies for sustainable mango cultivation.